Why should the gene be suggested to be inconstant when the contrary view, that gene structure is invariant except through mutagenic processes induced by potent external factors, has long been a universal doctrine of genetics? Indeed, during the early part of the present century before mutation was recognized as being of general occurrence, the seeming unvarying nature of the gene led to skepticism regarding the validity of the evolutionary theory; only later could the origins of the morphological differences between ...
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Why should the gene be suggested to be inconstant when the contrary view, that gene structure is invariant except through mutagenic processes induced by potent external factors, has long been a universal doctrine of genetics? Indeed, during the early part of the present century before mutation was recognized as being of general occurrence, the seeming unvarying nature of the gene led to skepticism regarding the validity of the evolutionary theory; only later could the origins of the morphological differences between individuals and species be attributed to a combination of mutation and evolution, involving natural forces selecting between favorable and unfavorable genetic changes. But during the past several decades, as knowledge of the macromolecular constitution of or- ganisms has increased to the point where even the primary structures of the genes themselves are being revealed on a routine basis, it has become increas- ingly difficult to ascribe all the resulting observations to ordinary mutagenesis and natural selection. Some more profound mechanism often seems to be present that influences both the constancy and inconstancy of the genes, an ap- paratus whose existence this study hopes to reveal. In seeking to demonstrate the universality of this mechanism, data are sought through the numerous activities of organisms of many types wherever gene action changes are manifest.
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Add this copy of The Inconstant Gene to cart. $51.65, new condition, Sold by Ingram Customer Returns Center rated 5.0 out of 5 stars, ships from NV, USA, published 2013 by Springer.
Add this copy of The Inconstant Gene to cart. $58.38, new condition, Sold by GreatBookPrices rated 4.0 out of 5 stars, ships from Columbia, MD, UNITED STATES, published 2013 by Springer.
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New. Trade paperback (US). Glued binding. 581 p. Contains: Unspecified, Illustrations, black & white. In Stock. 100% Money Back Guarantee. Brand New, Perfect Condition, allow 4-14 business days for standard shipping. To Alaska, Hawaii, U.S. protectorate, P.O. box, and APO/FPO addresses allow 4-28 business days for Standard shipping. No expedited shipping. All orders placed with expedited shipping will be cancelled. Over 3, 000, 000 happy customers.
Add this copy of The Inconstant Gene to cart. $81.67, good condition, Sold by Bonita rated 4.0 out of 5 stars, ships from Newport Coast, CA, UNITED STATES, published 2013 by Springer.
Add this copy of The Inconstant Gene (Nato Advanced Study Institutes to cart. $7.99, good condition, Sold by Wonder Book - Member ABAA/ILAB rated 5.0 out of 5 stars, ships from Frederick, MD, UNITED STATES, published 1983 by Springer.
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Good. Good condition. Good dust jacket. A copy that has been read but remains intact. May contain markings such as bookplates, stamps, limited notes and highlighting, or a few light stains.
Add this copy of The Inconstant Gene to cart. $10.00, good condition, Sold by Liberty Book Shop rated 4.0 out of 5 stars, ships from Avis, PA, UNITED STATES, published 1983 by Plenum Press..
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Very Good+ in Very Good+ dust jacket. 0306410842. In DJ. X-library w/internal & external marks Prepared for library, but never circulated.; HB, sm 4to; 581 pages.
Add this copy of The Inconstant Gene to cart. $41.27, very good condition, Sold by Cambridge Rare Books rated 4.0 out of 5 stars, ships from Cambridge, Gloucestershire, UNITED KINGDOM, published 1983 by Springer.
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1983. Plenum Press. American. Hard Cover. Book-VG. Dj-VG, minor edgewear. 9.5x6.5. 581pp. Some b/w photos. The author examines changes in gene expression that occur in cells during the course of their life cycles and postulates the existence of a 'supramolecular genetic mechanism' that could induce such changes.
